人体含违禁品液体标本检测试剂

人体含违禁品液体标本检测试剂

广州健仑生物科技有限公司

广州健仑生物科技有限公司是集研制开发、销售、服务于一体的优良企业,公司产品涉及临床快速诊断试剂、食品安全检测试剂，违禁品快速检测，动物疾病防疫检测试剂，免疫诊断试剂、临床血液学和体液学检验试剂、微生物检验试剂、分子生物学检验试剂、临床生化试剂、有机试剂等众多领域，同时核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名诊断产品集团公司产品，致力于为商检单位、疾病预防控制中心、海关出入境检疫局、卫生防疫单位,缉毒系统，戒毒中心，检验检疫单位、生化企业、科研院所、医疗机构等机构与行业提供*、高品质的产品服务。此外，本公司还开展食品、卫生、环境、药品等多方面的第三方检测服务。

广州健仑生物长期供应各种违禁品检测试纸、违禁品检测卡、违禁品检测试剂盒、药筛试纸、药筛试剂盒、吗啡检测试剂盒、巴比妥检测试剂盒等。

使用说明书

【产品名称】

通用名称：人体含违禁品液体标本检测试剂

英文名称：Diagnostic Kit for Nicotine（Colloidal Gold）

【包装规格】 

1人份/袋、40人份/盒

Multi DOA Screen Panel are rapid chromatographic immunoassays for the qualitative and simultaneous detection of one to fourteen of the following drugs in a variety of combinations in human urine. The designed cutoff concentrations and direct calibrator for these drugs are as follows:

The test is used to obtain visual qualitative result and is intended for health care professionals use including professionals at point of care sites to assist in the determination of drug compliance. It is not intended for over the counter sale to non-professionals.

This assay provided only a preliminary analytical test result. A more specific alternative chemical method must be used in order to obtain a confirmed analytical result. Gas Chromatography/ Mass Spectrometry (GC/MS) or Liquid Chromatography/ Mass Spectrometry (LC/MS) are the preferred confirmatory method. Clinical consideration and professional judgment should be applied to any drug of abuse test result, particularly when preliminary positive results are indicated.

本品采用竞争抑制法和胶体金免疫层析技术，用于定性检测人体尿液中尼古丁，适用于尼古丁药物滥用的初步筛查。

主营品牌：美国NovaBios、美国Cortez、国产创仑等等。

主要用途：筛查违禁品滥用残留、麻醉药残留、兴奋药物残留等等。

检测范围：吗啡、KET、mamp、MDMA、BZO、THC、巴比妥、MTD、BAR、MDMA、AMP、BUP、PCP、TCA、OXY、MET等等。

产品特点：可以根据需求自主订制多联卡。多联卡自由组合，从二联到十五联都可以订制。

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

如需订购或者了解请以下或

mob： 杨   ： 

美国NOVABIOS多联检测杯简介：

美国NOVABIOS单卡产品简介：

w the test device, urine specimen, and/or controls to reach room temperature (15-30oC) prior to t

更多产品说明可通过下方的进行了解

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【公司名称】 广州健仑生物科技有限公司

【 市场部 】       杨永汉
【】 
【腾讯  】
【公司地址】 广州市清华科技园健新基地番禺石楼镇健启路63号二期2幢101-103室

线粒体损  伤的另一种 常见改变为髓鞘样层状结构的形成，这是线粒体膜损  伤的结果。 衰亡或受损的线粒体，zui终由细胞的自噬过程加以处  理并zui后被 溶酶体酶所降解消化。线粒体是直接利用氧气制造能  量的部位， %以上吸入体内的氧气被线粒体消耗掉。但是，氧是个  “剑”， 一方面生物体利用氧分子制造能量，另一方面氧分子在  被利用的 过程中会产生极活泼的中间体（活性氧自由基）伤害生  物体造成 氧毒性。生物体就是在不断地与氧毒性进行斗争中求得  生存和发 展的，氧毒性的存在是生物体衰老的zui原初的原因。线  粒体利用 氧分子的同时也不断受到氧毒性的伤害，线粒体损伤超  过一定限 度，细胞就会衰老死亡。生物体总是不断有新的细胞取  代衰老的 细胞以维持生命的延续，这就是细胞的新陈代谢。线粒  体与疾病 编辑人类线粒体出现问题会导致线粒体病，线粒体病是  一大类遗 传代谢病，线粒体病主要包括：母系遗传Leigh综合征、  线粒体 肌病、多系统疾病、心肌病、进行性眼外肌麻痹、Leer遗  传性 视神经病、线粒体肌病，肌病，糖尿病和耳聋、共济失调舞  蹈病 、细胞外基质慢性游走性红斑、进行性眼外肌麻痹、肌红蛋  白尿 电机神经元疾病。
Another common change in mitochondrial damage is the formation of a myelin-like layered structure, which is the result of mitochondrial membrane damage. The dying or damaged mitochondria are eventually treated by the autophagic process of the cells and finally digested by lysosomal enzymes. Mitochondria are sites where oxygen is used directly to make energy, and more than % of the oxygen in the body is consumed by mitochondria. However, oxygen is a “sword”. On the one hand, organisms use oxygen molecules to make energy. On the other hand, when oxygen molecules are used, an extremely reactive intermediate (active oxygen radicals) is generated to cause harm to organisms and cause oxygen toxicity. Organisms continue to survive and thrive in the fight against oxygen toxicity. The presence of oxygen toxicity is the most primitive cause of organism aging. Mitochondria use oxygen molecules and continue to be harmed by oxygen toxicity. Mitochondrial damage exceeds a certain limit and cells die and die. Organisms always have new cells to replace aging cells to maintain the continuation of life, which is the cell's metabolism. Mitochondria and Disease Editing Human mitochondrial problems can cause mitochondrial diseases. Mitochondrial disease is a major category of inherited metabolic diseases. Mitochondrial disorders include maternal inherited Leigh syndrome, mitochondrial myopathy, multiple system diseases, cardiomyopathy, and progressive extraocular muscles. Paralysis, Leer's hereditary optic neuropathy, mitochondrial myopathy, myopathy, diabetes and deafness, ataxia chorea, extracellular matrix chronic migratory erythema, progressive extraocular muscle paralysis, myoglobinuria motor neuron disease