疟原虫抗体 荧光PCR检测试剂盒

疟原虫抗体 荧光PCR检测试剂盒

广州健仑生物科技有限公司

（广州健仑生物科技有限公司是集研制开发、销售、服务于一体的优良企业,公司产品涉及临床快速诊断试剂、食品安全检测试剂，违禁品快速检测，动物疾病防疫检测试剂，免疫诊断试剂、临床血液学和体液学检验试剂、微生物检验试剂、分子生物学检验试剂、临床生化试剂、有机试剂等众多领域，同时核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名诊断产品集团公司产品，致力于为商检单位、疾病预防控制中心、海关出入境检疫局、卫生防疫单位,缉毒系统，戒毒中心，检验检疫单位、生化企业、科研院所、医疗机构等机构与行业提供*、高品质的产品服务。此外，本公司还开展食品、卫生、环境、药品等多方面的第三方检测服务。）

疟原虫抗体 荧光PCR检测试剂盒 本试剂盒主要是采用胶体金层析的原理制成，用于检测人体血清/血浆/全血标本中，感染的疟原虫抗体，包括了恶性疟原虫和间日疟原虫、卵形疟原虫、三日疟原虫共有抗原的鉴别性检测。

人群易感性 人群对疟疾普遍易感，感染后虽有一定的免疫力，但不持久，各型疟疾之间亦无交叉免疫性，经反复多次感染后，再感染时症状可较轻，甚至无症状，而一般非流行区来的外来人员常较易感染，且症状较重。
People susceptible to the crowd generally susceptible to malaria, although the infection after a certain degree of immunity, but not lasting, there is no cross-immunity between malaria, after repeated infections, re-infection symptoms may be lighter, or even Asymptomatic, while the non-endemic areas of non-migrant workers are often more susceptible to infection, and the symptoms are severe.

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1 撕开检测卡铝箔袋，取出袋内金标卡。注意：不要让袋内材料暴露于高温高湿环境，撕开铝箔袋后尽快使用。

2将金标卡平放在台面上；并将病人名字和编号写在标签上。

3 取5微升(吸管*刻度处)全血标本，垂直加入金标卡上“加样孔A”内。

4 掰断裂解液瓶子盖子上方的绿色圆头，在“样品孔B”上垂直滴加4滴裂解液。

5 在十五分钟内出结果。注意：必须在15分钟内判读结果，如超时判断，结果无效。

6 请遵循相关法规，妥善处理样本及废弃材料。

7 存储条件：2-30℃；

8 保质期：18个月；

【病原学检测】

疟疾检测,用于检测出虐疾的病原体——疟原虫，是明确诊断的zui直接证据。目前常用的层析法，具有操作简单、灵敏度高和可鉴别虫种等优点，广泛用于疟疾的病原学诊断，是目前zui常用的方法之一。

我司为美国NOVABIOS公司在中国地区战略合作伙伴，负责该公司产品的总经销及售后服务工作。还与各疾控中心，疾病防御中心有合作关系，例如中国疾病预防控制中心 、浙江省疾病预防控制中心  ，详情可以我司工作人员。

（  MOB：杨永汉）  

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

广州健仑生物长期供应各种违禁品检测试纸、违禁品检测卡、违禁品检测试剂盒、药筛试纸、药筛试剂盒、吗啡检测试剂盒、巴比妥检测试剂盒等。

想了解更多的产品及服务请扫描下方二维码：

【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

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【腾讯  】 
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103

2.培养特性
各种螺旋体在生理上的需求不一样，能量来源于糖类、氨基酸和长链脂肪酸类等。
3.抗原成分
大部分螺旋体的抗原成分主要有外膜蛋白和内鞭毛抗原，其他抗原成分因菌而异。
4.抵抗力
螺旋体的抵抗力较弱，但钩端螺旋体的抵抗力强于梅毒螺旋体。螺旋体的致病机制尚不明了。不同的螺旋体通过不同的致病物质和机制而致病。钩端螺旋体致病可能与其具有溶解红细胞的溶血素、细胞毒因子及内毒素样物质有关。梅毒螺旋体的致病机制与其表面夹膜样物质的黏附作用及其分泌的黏多糖酶作用有关。螺旋体的免疫性质多以体液免疫为主。钩端螺旋体病*后可获得对同型钩体的持久体液免疫。梅毒螺旋体的免疫是传染性免疫，受染机体产生的特异性抗体可介导杀死或溶解梅毒螺旋体。有较密切的结核病接触史，起病可急可缓，多为低热（午后为著）、盗汗、乏力、纳差、消瘦、女性月经失调等；呼吸道症状有咳嗽、咳痰、咯血、胸痛、不同程度胸闷或呼吸困难。
2.体征
肺部体征依病情轻重、病变范围不同而有差异，早期、小范围的结核不易查到阳性体征，病变范围较广者叩诊呈浊音，语颤增强，肺泡呼吸音低和湿啰音。晚期结核形成纤维化，局部收缩使胸膜塌陷和纵隔移位。在结核性胸膜炎者早期有胸膜摩擦音，形成大量胸腔积液时，胸壁饱满，叩诊浊实，语颤和呼吸音减低或消失。
3.肺结核的分型和分期
（1）肺结核分型 ①原发型肺结核（Ⅰ型） 肺内渗出病变、淋巴管炎和肺门淋巴结肿大的哑铃状改变的原发综合征，儿童多见，或仅表现为肺门和纵隔淋巴结肿大。②血行播散型肺结核（Ⅱ型） 包括急性粟粒性肺结核和慢性或亚急性血行播散型肺结核两型。急性粟粒型肺结核：两肺散在的粟粒大小的细菌影，大小*密度相等，分布均匀的粟粒状细菌影，随病期进展，可互相融合；慢性或亚急性血行播散型肺结核：两肺出现大小不一、新旧病变不同，分布不均匀，边缘模糊或锐利的结节和索条细菌影。③继发型肺结核（Ⅲ型） 本型中包括病变以增殖为主、浸润病变为主、干酪病变为主或空洞为主的多种改变。
2. C*tion characteristics
Various spirochetes in the physiological needs are not the same, the energy comes from carbohydrates, amino acids and long-chain fatty acids and so on.
Antigen composition
Most of the antigen components of spirochetes are mainly outer membrane proteins and internal flagellar antigens, other antigen components vary with bacteria.
4 resistance
The resistance of spirochetes is weak, but the resistance of leptospira is stronger than that of Treponema pallidum. Pathogenesis of spirochetes is not yet clear. Different spirochetes cause disease through different pathogenic agents and mechanisms. Leptospirosis may be related to its hemolysin with red blood cells, cytotoxic agents and endotoxin-like substances. Pathogenesis of Treponema pallidum and its surface membrane-like substance adhesion and secretion of mucolytic enzyme role. The immune nature of spirochetes is dominated by humoral immunity. After leptospirosis is recovered, a sustained humoral immunity to the leptospira can be obtained. Treponema pallidum immunization is contagious immunity, the infected body produces specific antibodies can mediate killing or dissolving Treponema pallidum. There is a closer contact history of tuberculosis, the onset of emergency can be slow, mostly low fever (for the afternoon), night sweats, fatigue, anorexia, weight loss, menstrual disorders, etc .; respiratory symptoms are cough, sputum, hemoptysis, chest pain, Different degrees of chest tightness or breathing difficulties.
2. signs
Pulmonary signs according to the severity of the disease vary in different areas, early, small tuberculosis is not easy to find positive signs, lesions were extensive percussion was voiced, increased trembling, alveolar breath sounds low and wet rales. Advanced tuberculosis fibrosis, local contraction of the collapse of the pleura and mediastinal shift. Tuberculous pleurisy in early pleural friction, the formation of a large number of pleural effusion, the chest wall full, percussion turbidity, fibrillation and breath sounds reduce or disappear.
Tuberculosis classification and staging
(1) tuberculosis classification ① primary pulmonary tuberculosis (type Ⅰ) intrapulmonary exudative lesions, lymphangitis and hilar lymph nodes enlarged dumbbell-shaped primary syndrome, more common in children, or only as hilar And mediastinal lymph nodes. ② hematogenous disseminated tuberculosis (type Ⅱ), including acute miliary tuberculosis and chronic or subacute hematogenous disseminated tuberculosis two types. Acute miliary tuberculosis: mildew-sized bacterial shadow scattered in both lungs, uniform and uniform density of the size of uniform miliary bacterial shadow, with the progression of the disease, can be mutually integrated; chronic or subacute hematogenous disseminated tuberculosis: two lungs appear Different sizes, different old and new lesions, uneven distribution, blurred edges or sharp nodules and cable bacteria shadow. ③ secondary pulmonary tuberculosis (type Ⅲ) This type includes lesions mainly proliferation, infiltration of lesions, mainly cheese lesions or empty-based variety of changes.