DDIT3（12q13）基因断裂探针
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DDIT3（12q13）基因断裂探针 ERG是母红血球病病毒E26转换序列（ETS）转录因子的基因家族成员之一，它在淋巴细胞和内皮细胞表达，调节内皮细胞凋亡和血管生成。研究发现ERG在良性和恶性的血管肿瘤都有表达。用于检测甲状腺滤泡癌常见的PPARγ基因断裂及与其他基因的易位。 本试剂盒仅供科研使用。
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DDIT3（12q13）基因断裂探针
广州健仑生物科技有限公司
本司长期供应尼古丁（可替宁）检测试剂盒，其主要品牌包括美国NovaBios、广州健仑、广州创仑等进口产品，国产产品，试剂盒的实验方法是胶体金方法。
我司还有很多荧光原位杂交系列检测试剂盒以及各种FISH基因探针和染色体探针等，。
DDIT3（12q13）基因断裂探针
用于检测DDIT3（12q13）基因断裂的情况。
本试剂盒仅供科研使用。
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以下是我司出售的部分FISH产品：
| FOXR2(Xp11)基因断裂探针 |
| 6q27(MLLT4)检测探针 |
| NUT(15q14)检测探针 |
| CDKN2A（9p21）基因探针（含9号着丝粒） |
| EWSR1/WT1融合基因t (11;22)探针 |
| [BCR/ABL(DF)]/ASS探针 |
| [BCRABL(DF)]/[ETV6/RUNX1]/KMT2A/IGH探针 |
| [PML/RARA]/CBFB/[AML1/ETO]/KMT2A探针 |
| ATM/p53/13q14/RB1/CSP12探针 |
| BCL6/MYC/[CCND1/IGH]/[BCL2/IGH]探针 |
| 20q/+8/[-7/-7q]/[-5/-5q]/-Y探针 |
| CDK4（12q14）基因扩增探针 |
| WWTR1/CAMTA1融合基因t (1;3)探针 |
| MGEA5（10q24）基因断裂探针 |
| D13S319基因探针（红色） |
| HMGA2（12q14）基因断裂探针 |
| MYB/NFIB融合基因t(6;9)探针 |
| FOSB（19q13）基因断裂探针 |
我司还提供：登革热，黄热病，基肯孔热，西尼罗河，立次克体，无形体，蜱虫，恙虫，锥虫，利什曼原虫，RK39, 汉坦病毒，乙脑，森林脑炎，寨卡病毒 ，H7N9 ,流感，霍乱，军团菌，结核，诺如病毒，轮状病毒，炭疽，O157，葡萄球菌 ，流行性出血热，伤寒杆菌，志贺氏菌检测试剂,！
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【公司名称】 广州健仑生物科技有限公司
【】 杨永汉
【】
【腾讯 】
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-3室
【企业文化宣传】
After 6 years of exploration, Shanghai experts found genetic characteristics and genetic background of China patients with esophageal squamous cell carcinoma, and for the first time that an important cause of such China Asian population at high risk of esophageal squamous cell carcinoma NFE2L2 gene "germline mutation" risk higher than others.
In January 4th, reporters from the Tumour Hospital Affiliated to Fudan University (www.thepaper。。ｃｎ) received the news from Tumour Hospital Affiliated to Fudan University. The research results are also published online in the Nature Communications, a sub issue of the international authoritative journal Nature.
Find the "culprit" in the difference of genetic map
China is a country with high incidence of esophageal cancer. The number of cases is 50% of the world, of which 90% is squamous cell carcinoma. In the United States, the incidence of esophageal cancer is low, and the pathological type is mainly adenocarcinoma.
In addition to the effects of living habits and carcinogens, what are the reasons for the incidence and pathological types of esophageal cancer, with such a large demographic difference?
Professor Zhao Kuaile tried to analyze the genetic material DNA in human cells from the source, and then explore the genetic difference of esophageal cancer in different races. "If we can find the pathogenesis of esophageal cancer from the gene level, we will provide a new strategy basis for the prevention and treatment of esophageal cancer for Chinese race," Zhao Kuaile said.
