果氏巴贝西虫IgG免疫荧光试剂盒

果氏巴贝西虫IgG免疫荧光试剂盒

Babesia microti IgG IFA Kit

广州健仑生物科技有限公司

主要用途：用于检测人血清中的果氏巴贝西虫IgG抗体

产品规格：12 孔/张,10 张/盒

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果氏巴贝西虫IgG免疫荧光试剂盒

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【公司名称】 广州健仑生物科技有限公司
【】    杨永汉 
【】 
【腾讯 】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-3室
【企业文化】

然而，加州大学圣地亚哥分校医学院和穆尔斯癌症中心的研究人员发现，调节抗原抗体期间的干细胞活化的信号被癌细胞劫持，用来生产快速增长，更具侵略性的肿瘤。相关文章发表于2014年8月11日的《Developmental Cell》杂志上。
Cell子刊:癌细胞劫持抗原抗体干细胞活化信号来增殖的机制
抗原抗体与抗原抗体癌之间的早已知道，但抗原抗体和抗原抗体癌风险之间的关系却很复杂。虽然有一个孩子会降低妇女在以后生活中患抗原抗体癌的风险，但是，每次抗原抗体后都会有一个抗原抗体癌的高侵袭性形式发展的短期风险增加。目前的研究表明，怀孕期间，影响干细胞行为的重要分子可能促进了这些与抗原抗体相关的更具侵袭性抗原抗体癌的发展，研究人员计划进一步研究这一种可能性。
作者们很快指出，他们的研究结果不应该作为一个抗原抗体的理由。这个信号通路被癌细胞劫持并不能作为一个触发抗原抗体癌的原因。相反，它可能恶化或加速由其他因素引发的癌症，如潜在突变或遗传倾向。
“我们的研究并没有指出，触发癌症的真正原因，而是解释了，一旦癌症启动会发生什么，”病理学特聘教授和研发副主席David A. Cheresh博士说。“这里有一个比喻：得癌症，你首先要开始激活一个致癌基因，一个携带突变并能够启动癌症的基因。这个致癌基因相当于汽车的点火开关，而被癌细胞利用的信号通路就像汽油。有了它，汽车才能启动，这也意味着如果致癌基因还没有开始激活，那么癌症就不会发展。”
在本研究中，研究人员集中于一个被称为整合素的细胞表面受体蛋白家族，它们充当关键的通信渠道，zui终在这家称为β-3整合的一个成员的作用归零。也被称为CD61，它已经与转移和抗肿瘤的药物有关。
作者指出，CD61可以作为显示参与怀孕和癌症期间抗原抗体发育的信号通路的良好标志。它很容易被检测到，并可以用来诊断和治疗乳房癌病例。

However, researchers at the University of California San Diego School of Medicine and the Moores Cancer Center found that signals that regulate stem cell activation during antigen-antibody abstraction were hijacked by cancer cells to produce rapidly-growing, more aggressive tumors. Related article was published in Developmental Cell magazine on August 11, 2014.
Cell Subcategory: Mechanisms of Proliferation of Cancer Cells Hijacking Antigen-derived Antibody Stem Cells
The link between antigen and antigen-antibody cancers has long been known, but the relationship between antigen-antibody and antigen-antibody cancer risk is complex. Although having one child reduces the risk of developing anti-cancer antibodies in later life, there is an increased short-term risk of development of a highly aggressive form of antigen-antibody cancer after each antigen-antibody. Current research shows that important molecules that influence stem cell behavior during pregnancy may contribute to the development of more aggressive antigen-antibody cancers associated with antigen antibodies, and the researchers plan to investigate this possibility further.
The authors quickly pointed out that their findings should not be used as a reason for antigen-antibody. This signal pathway is hijacked by cancer cells and does not serve as a trigger for antigen-antibody cancer. On the contrary, it may exacerbate or accelerate cancer caused by other factors, such as potential mutations or genetic predisposition.
"Our research did not point out the real cause of triggering cancer but explained what would happen once the cancer started," said David A. Cheresh, Ph.D., professor of pathology and vice chairman of R & D. Here's a metaphor: In cancer, you first have to start activating an oncogene, a gene that carries the mutation and activates the cancer, an oncogene that's equivalent to a car's ignition switch, and the signaling pathways used by cancer cells are like petrol. With it, the car can start, which means that if the oncogenes have not yet started to activate, then the cancer will not develop. "
In this study, researchers focused on a family of cell-surface receptor proteins called integrins that served as key communication channels and eventually zeroed in the role of a member of the family called beta-3 integration. Also known as CD61, it has been associated with metastatic and anti-tumor drugs.
The authors note that CD61 can serve as a good indicator of the signaling pathways involved in antigen and antibody development during pregnancy and cancer. It is easily detectable and can be used to diagnose and treat cases of breast cancer.