编号	英文名称	中文名称
JL-FA-01	Amebiasis (AME)	阿米巴病
JL-FA-02	Allergens, Rast scores	过敏原，放射性过敏原吸收实验。指对特定的人群引起免疫反应或者过敏反应的食品中的蛋白质
JL-FA-03	Allergens, Rast scores negative	过敏原，放射性过敏原吸收实验阴性
JL-FA-04	Anti-cyclic citrullinated peptide Antibody (CCP) Arthritis	抗环瓜氨酸肽抗体
JL-FA-05	ASCA Saccharomyces Cerevi	人抗酿酒酵母抗体（ASCA）
JL-FA-06	Aspergillis	麴菌病
JL-FA-07	Beta 2 Glycoprotein	β2糖蛋白
JL-FA-08	Beta 2 Glycoprotein IgM	β2糖蛋白 IGM
JL-FA-09	Bordela Pertussis	百日咳杆菌
JL-FA-10	Bordela Pertussis IgM	百日咳杆菌 IGM
JL-FA-11	C-ANCA	C-抗中性粒细胞胞浆抗体（ANCA)
JL-FA-12	Cardiolipin	心肌磷脂
JL-FA-13	Cardiolipin IgA	心肌磷脂 IGA
JL-FA-14	Cardiolipin IgG	心肌磷脂 IGG
JL-FA-15	Cardiolipin IgM	心肌磷脂 IGM
JL-FA-16	Cerebral Spinal Fluid	脑脊髓液
JL-FA-17	Chagas	恰加斯病/南美锥虫
JL-FA-18	Chlamydia	衣原体
JL-FA-19	Chlamydia IgA	衣原体IGA
JL-FA-20	Chlamydia IgG	衣原体IGG
JL-FA-21	Chlamydia IgM	衣原体IGM
JL-FA-22	Chlamydia Neg	衣原体阴性
JL-FA-23	Clotting Factor C3	凝固因子C3
JL-FA-24	Clotting Factor C4	凝固因子C4
JL-FA-25	Coccidiodes	球孢菌
JL-FA-26	Cytomegalovirus (CMV) Neg	巨细胞病毒抗体阴性
JL-FA-27	CMV IgG	巨细胞病毒 IGG阳性
JL-FA-28	CMV IgM VCA	巨细胞病毒 IGM 阳性
JL-FA-29	C-Reactive Protein (CRP)	C-反应蛋白质
JL-FA-30	Dengue Fever	登革热
JL-FA-31	Dengue Fever IgM	登革热 IGM
JL-FA-32	DS (Double Stranded) DNA	双链脱氧核糖核酸
JL-FA-33	EBNA (Epstein-Barr nuclear antigen) IgG	EB病毒核抗原 IGG
JL-FA-34	EBNA (Epstein-Barr nuclear antigen) IgM	EB病毒核抗原 IGM
JL-FA-35	Epstein Barr Virus (EBV) Negative Plasma	EB病毒阴性血浆
JL-FA-36	Epstein Barr Virus (EBV) EA IgM	EB病毒早期抗原 IGM
JL-FA-37	Epstein Barr Virus (EBV) VCA IgM	EB病毒壳蛋白 IGM
JL-FA-38	Epstein Barr Virus (EBV) EA IgG	EB病毒早期抗原 IGG
JL-FA-39	EMA (Endomysial Antibodies)	肌内膜
JL-FA-40	Gliadin	麸蛋白,麦醇溶蛋白,麦胶蛋白
JL-FA-41	Gliadin IgG	麦醇溶蛋白 IGG
JL-FA-42	Gliadin IgA	麦醇溶蛋白 IGA
JL-FA-43	Glomerular Basement Membrane (GBMA)	肾小球基底膜病
JL-FA-44	Helicobacter pylori IgA	幽门螺旋杆菌IGA
JL-FA-45	Helicobacter pylori IgG	幽门螺旋杆菌IGG
JL-FA-46	Helicobacter pylori IgM	幽门螺旋杆菌IGM
JL-FA-47	Helicobacter pylori Negative	幽门螺旋杆菌阴性
JL-FA-48	Helicobacter pylori Positive Plasma	幽门螺旋杆菌阴性血浆
JL-FA-49	Hepatitis A Virus (HAV) Pos. Plasma	甲型肝炎病毒阳性血浆
JL-FA-50	Hepatitis A Virus (HAV) IgM	甲型肝炎病毒IGM
JL-FA-51	Hepatitis B Core (HBc) IgG	乙型肝炎病毒核心 IGG
JL-FA-52	Hepatitis B Core (HBc) IgM	乙型肝炎病毒核心 IGM
JL-FA-53	Anti Hbe (Antibody to HBV antigen)	乙肝抗体
JL-FA-54	Hepatitis Delta Virus	丁型肝炎病毒
JL-FA-55	HBeAg (HBV e antigen)	乙肝 E抗原
JL-FA-56	anti-HBs (HBV surface antibody)	乙肝表面抗体
JL-FA-57	Hepatitis B (HBsAg) "Chronic"	乙型肝炎（乙肝表面抗原）“慢性病
JL-FA-58	HBsAg (HBV surface antigen) Serum	乙肝表面抗原血清
JL-FA-59	HBsAg (AD)	乙肝表面抗原（AD）
JL-FA-60	HBsAg (AY)	乙肝表面抗原（AY）
JL-FA-61	HBV Positive Plasma	乙肝阳性血浆
JL-FA-62	HBV DNA Plasma	乙肝DNA血浆
JL-FA-63	HBV DNA Serum	乙肝DNA血清
JL-FA-64	HBV DNA type A	A型乙肝DNA
JL-FA-65	HBV DNA type B	B型乙肝DNA
JL-FA-66	HBV DNA type C	C型乙肝DNA
JL-FA-67	HBV DNA type D	D型乙肝DNA
JL-FA-68	HBV DNA type E	E型乙肝DNA
JL-FA-69	HBV DNA type F	F型乙肝DNA
JL-FA-70	HBV Antibody HCV Antibody Plasma CO-INFECTED	乙肝和丙肝联合感染血浆
JL-FA-71	HCV (Hepatitis C Virus) Antibody	丙型肝炎抗体
JL-FA-72	HCV Core Antigen Positive	丙肝核心抗原阳性
JL-FA-73	HCV RNA PLASMA Genotype 1	基因1型丙肝RNA 血浆
JL-FA-74	HCV RNA PLASMA Genotype 2	基因2型丙肝RNA 血浆
JL-FA-75	HCV RNA PLASMA Genotype 3	基因3型丙肝RNA 血浆
JL-FA-76	HCV RNA PLASMA Genotype 4	基因4型丙肝RNA 血浆
JL-FA-77	HCV RNA PLASMA Genotype 5	基因5型丙肝RNA 血浆
JL-FA-78	HCV RNA PLASMA Genotype 6	基因6型丙肝RNA 血浆
JL-FA-79	HCV Riba single band	丙肝免疫印迹单波段
JL-FA-80	HCV RIBA Pos. (multiple bands)	丙肝免疫印迹阳性多波段
JL-FA-81	HCV Negative	丙肝阴性
JL-FA-82	HCV RNA Pos (quantitative)	丙肝RNA阳性（定量）
JL-FA-83	Hepatitis E	戊型肝炎
JL-FA-84	Herpes Simplex Virus (HSV)1/2 Positive Plasma	单纯性疱疹病毒1/2阳性血浆
JL-FA-85	Herpes Simplex Virus (HSV) 1 Negative Plasma	单纯性疱疹病毒1 阴性血浆
JL-FA-86	Herpes Simplex Virus (HSV) 1 IgG	单纯性疱疹病毒1 IGG
JL-FA-87	Herpes Simplex Virus (HSV 1) IgM	单纯性疱疹病毒1 IGM
JL-FA-88	Herpes Simplex Virus (HSV) 2 IgG	单纯性疱疹病毒2 IGG
JL-FA-89	Herpes Simplex Virus (HSV) 2 IgM	单纯性疱疹病毒2 IGG
JL-FA-90	Histone	组蛋白
JL-FA-91	Human Anti Mouse Ab (HAMA)	人抗鼠抗体
JL-FA-92	Human immunodeficiency virus (HIV) 1 Neg	HIV I 阴性
JL-FA-93	anti Human immunodeficiency virus (HIV) 1 Plasma	抗HIV I 血浆
JL-FA-94	anti Human immunodeficiency virus (HIV) 1 Serum	抗HIV I 血清
JL-FA-95	anti Human immunodeficiency virus (HIV) 2 Western Blot Tested	抗HIV 2 免疫印迹
JL-FA-96	anti Human immunodeficiency virus (HIV) 1/2 2 HIV (+)	抗HIV 1/2 2 HIV阳性
JL-FA-97	Human immunodeficiency virus (HIV) Ag	HIV抗原
JL-FA-98	HIV RNA (quantitative) Plasma	HIV RNA 定量血浆
JL-FA-99	HIV RNA (quantitative) Serum	HIV RNA 定量血清
JL-FA-100	HIV1 Subtype A	HIV1 亚型A
JL-FA-101	HIV1 Subtype B	HIV1 亚型B
JL-FA-102	HIV1 Subtype C	HIV1 亚型C
JL-FA-103	HIV1 Subtype D	HIV1 亚型D
JL-FA-104	HIV1 Subtype E	HIV1 亚型E
JL-FA-105	HIV1 Subtype F	HIV1 亚型F
JL-FA-106	HIV1 Subtype G	HIV1 亚型G
JL-FA-107	HIV1 Subtype H	HIV1 亚型H
JL-FA-108	HIV1 Subtype J	HIV1 亚型J
JL-FA-109	HIV1 Subtype K	HIV1 亚型K
JL-FA-110	HIV1 Group O	HIV1 亚型O
JL-FA-111	Human immunodeficiency virus (HIV) 2 Antibody Plasma	HIV 2 抗体血浆
JL-FA-112	Human immunodeficiency virus (HIV) 2 Antibody Serum	HIV 2 抗体血清
JL-FA-113	HPV (Human Papiloma Virus) Negative	人乳状瘤病毒HPV阴性
JL-FA-114	HPV (Human Papiloma Virus) Positive	人乳状瘤病毒HPV阳性
JL-FA-115	Human immunodeficiency virus (HIV) Antibody HCV Antibody Plasma COINFECTED	HIV 抗体 HCV
JL-FA-116	Human T-cell Lymphotropic Virus (HTLV) I/II	人嗜T淋巴细胞病毒(HTLV) I/II
JL-FA-117	Human T-cell Lymphotropic Virus (HTLV) I	人嗜T淋巴细胞病毒(HTLV) I
JL-FA-118	Human T-cell Lymphotropic Virus (HTLV) II	人嗜T淋巴细胞病毒(HTLV) II
JL-FA-119	Jo-1	多发性肌炎抗原JO-1
JL-FA-120	IgE < 5,000 Ku/L	IgE < 5,000 Ku/L
JL-FA-121	Legionella	军团杆菌属
JL-FA-122	Leptospira	军团杆菌属
JL-FA-123	Lyme Disease	莱姆(氏)病:蜱传播的全身性疾病,常在夏季发生
JL-FA-124	Lyme IgG	莱姆(氏)病 IGG
JL-FA-125	Lyme IgM	莱姆(氏)病 IGM
JL-FA-126	Lyme Disease Neg	莱姆(氏)病阴性
JL-FA-127	Malaria	疟疾
JL-FA-128	Mononucleosis (infectious)	单核细胞增多症（有传染性的）
JL-FA-129	Mononucleosis Negative	单核细胞增多症阴性
JL-FA-130	Measles Negative	麻疹阴性
JL-FA-131	Measles IgG	麻疹 IGG
JL-FA-132	Measles IgM	麻疹 IGM
JL-FA-133	Microsomal Anti-thyroid peroxidase antibody (TPO) Positive Plasma Standard Titer (typically 1,000-3,000 IU/mL)	微粒体抗甲状腺过氧化物酶抗体
JL-FA-134	Microsomal Anti-thyroid peroxidase antibody (TPO) Negative Plasma	微粒体抗甲状腺过氧化物酶抗体
JL-FA-135	Anti-mitochondrial antibody (AMA)	抗线粒体抗体
JL-FA-136	Multiple Sclerosis	多发性硬化症
JL-FA-137	Mumps IgG	流行性腮腺炎 IGG
JL-FA-138	Mumps Ab IgM	流行性腮腺炎抗体 IGM
JL-FA-139	Mumps Antibody Negative Plasma	流行性腮腺炎抗体阴性血浆
JL-FA-140	Mumps Antibody Negative Serum	流行性腮腺炎抗体阴性血清
JL-FA-141	Myeloma Plasma	骨髓瘤血浆
JL-FA-142	Myeloma IgA	骨髓瘤IGA
JL-FA-143	Myeloma IgE	骨髓瘤IGE
JL-FA-144	Myeloma IgG	骨髓瘤IGG
JL-FA-145	Myeloma IgM	骨髓瘤IGM
JL-FA-146	Mycoplasma	支原体
JL-FA-147	Mycoplasma Negative	支原体阴性
JL-FA-148	Mycoplasma IgG	支原体IGG
JL-FA-149	Mycoplasma IgM	支原体IGM
JL-FA-150	Mycoplasma PCR	支原体PCR
JL-FA-151	Normal Human Plasma	正常人血浆
JL-FA-152	Normal Human Serum	正常人血清
JL-FA-153	Nuclear Antibody Centromere	核抗体着丝粒
JL-FA-154	Nuclear Antibody, Speckled ANA	核抗体，斑点抗核抗体
JL-FA-155	Nuclear Antibody, Nucleolar ANA	核抗体,核仁抗核抗体
JL-FA-156	Nuclear Antibody, Homogeneous ANA	核抗体，同质抗核抗体
JL-FA-157	Nuclear Antiobody, Speckled. (ANA) Negative	核抗体，斑点。抗核抗体阴性
JL-FA-158	P-ANCA (associated neutrophil cytoplasmic antibodies)	相关的嗜中性粒细胞胞浆抗体
JL-FA-159	Parietal Cell Antibody (PCA)	胃)壁细胞抗体
JL-FA-160	Parvo positive plasma	细小病毒阳性血浆
JL-FA-161	Parvo IgM	细小病毒 IGM
JL-FA-162	Parvo IgG	细小病毒 IGG
JL-FA-163	Parvo Negative Plasma	细小病毒阴性血浆
JL-FA-164	Parvo DNA positive	细小病毒 DNA 阳性
JL-FA-165	Phospholipid Positive Plasma	磷脂阳性血浆
JL-FA-166	Prothrombin	凝血酶原,凝血因子
JL-FA-167	Rheumatoid Factor (RF) <1000 IU/mL	类风湿因子<1000 IU/mL
JL-FA-168	Rheumatoid Factor (RF) 1001-2000 IU/mL	类风湿因子1001-2000 IU/mL
JL-FA-169	Rheumatoid Factor (RF) 2001-4000 IU/mL	类风湿因子 2001-4000 IU/mL
JL-FA-170	Rheumatoid Factor (RF) 4001-5000 IU/mL	类风湿因子 4001-5000 IU/mL
JL-FA-171	Rheumatoid Factor (RF) >5000 IU/mL	类风湿因子>5000 IU/mL
JL-FA-172	Ribonucleoprotein (RNP) Positive	核糖核蛋白阳性
JL-FA-173	Rubella Chimeric	风疹
JL-FA-174	Rubella Negative	风疹阴性
JL-FA-175	Rubella IgG	风疹IGG
JL-FA-176	Rubella IgM	风疹IGM
JL-FA-177	Rubeola Negative Plasma	风疹阴性血浆
JL-FA-178	Rubeola IgG	风疹IGG
JL-FA-179	Scleroderma (Scl-70) Pos	胶原沉着病,硬皮病,硬皮症阳性
JL-FA-180	Scleroderma (Scl-70) Negative	硬皮病阴性
JL-FA-181	Sickle Cell Fresh Whole Blood	镰刀形红细胞新鲜全血
JL-FA-182	Smith (SM)	抗Smith抗体阳性血清（SLE的特征性抗体）
JL-FA-183	SMITH RNP	抗RNP抗体阳性血清（SLE的特征性抗体）
JL-FA-184	Smooth Muscle (ASMA)	抗平滑肌抗体阳性血清
JL-FA-185	Sjogren syndrome antigen A (SSA) Positive	舍格伦综合征或干燥综合征抗原A 阳性
JL-FA-186	Sjogren syndrome antigen B (SSB) Positive	舍格伦综合征抗原B 阳性
JL-FA-187	Sjogren syndrome antigen B (SSB) Negative	舍格伦综合征抗原B阴性
JL-FA-188	Streptolysin O Ab (ASO)	链球菌溶血素O抗体
JL-FA-189	Syphilis (RPR - Rapid Plasma Reagin) Positive Plasma	梅毒（梅毒-快速血浆反应）阳性血浆
JL-FA-190	Syphilis (RPR - Rapid Plasma Reagin) Negative Plasma	梅毒（梅毒-快速血浆反应）阴性血浆
JL-FA-191	Syphilis/ATA/T. pallidum IgG	梅毒ATA/T，苍白球IGG
JL-FA-192	Syphilis/ATA/T. pallidum IgM	梅毒ATA/T，苍白球IGM
JL-FA-193	Systemic Lupus Erythematosus (SLE) Positive	全身性红斑狼疮阳性
JL-FA-194	Systemic Lupus Erythematosus (SLE) Negative	全身性红斑狼疮阴性
JL-FA-195	TG/TPO Positive (Standard Titer 1,000 - 3000 IU/mL)	甲状腺球蛋白/甲状腺过氧化物酶阳性
JL-FA-196	TG/TPO Negative	甲状腺球蛋白/甲状腺过氧化物酶阴性
JL-FA-197	TTG (Tissue Transglutaminase)	组织转谷氨酰胺酶
JL-FA-198	TTG (Tissue Transglutaminase) IgA	组织转谷氨酰胺酶 IGA
JL-FA-199	ToRCH (Toxo, Rubella, CMV, HSV) Positive	优生优育（弓形虫，风疹，巨细胞，单胞）阳性
JL-FA-200	ToRCH (Toxo, Rubella, CMV, HSV) Negative	优生优育（弓形虫，风疹，巨细胞，单胞）阴性
JL-FA-201	Toxoplasmosis (Toxo)	弓形虫病
JL-FA-202	Toxoplasmosis (Toxo) IgG	弓形虫病IGG
JL-FA-203	Toxoplasmosis (Toxo) IgM	弓形虫病IGM
JL-FA-204	Thyroglobulin (TG) Positive Plasma	甲状腺球蛋白阳性血浆
JL-FA-205	Thyroglobulin (TG) Negative	甲状腺球蛋白阴性
JL-FA-206	Varicella-Zoster Virus (VZV) Negative	水痘-带状疱疹病毒阴性
JL-FA-207	Varicella-Zoster Virus (VZV) IgG	水痘-带状疱疹病毒IGG
JL-FA-208	Varicella-Zoster Virus (VZV) IgM	水痘-带状疱疹病毒IGM
JL-FA-209	West Nile Virus (WNV)	西尼罗河脑炎病毒
JL-FA-210	West Nile Virus (WNV) IgM	西尼罗河脑炎病毒IGM

美国

此外，实验过程中还采用PET/CT进行扫描，测量棕色脂肪、肌肉、脂肪检测代谢变化。实验结果显示，棕色脂肪在冷的月份增加，在温暖的月份则会下滑。
棕色脂肪量增加能够影响代谢，其中一大好处就是提升胰岛素的敏感性——这意味着，体内棕色脂肪较多的人，餐后降低血糖水平只需要较少的胰岛素。
“在这项研究以前，是否能够实际控制人体棕色脂肪生长还是未知数。我们的研究发现，寒冷月份会让棕色脂肪增加30%至40%。而在第二个温度为24摄氏度的实验月份，棕色脂肪水平回落到基线水平。第四月——温度高达27度，棕色脂肪的量回落到基准线以下。”
在保罗-李博士看来，“胰岛素敏感性提升与棕色脂肪的增加同步，在未来，这或许能开启新的治疗方法，阻碍糖分代谢。另一方面，由于当代社会供热上升，人们在轻度寒冷的条件下暴露的时间减短，这可能损害棕色脂肪的功能，造成肥胖、代谢紊乱。”
“研究显示，过去几十年中在英国、美国家庭卧室、餐厅、客厅的温度从19摄氏度攀升至22摄氏度，这也将导致棕色脂肪生成减弱。因此，除了不健康的饮食、缺乏运动之外，在较高温度下暴露时间增长，也可能是导致肥胖的因素之一。”
蝾螈成功再生身体部位的秘密正在被揭开，伦敦大学学院的研究人员为了将其应用于人类。首先，研究人员发现‘ERK通路’必须不断地活动为蝾螈细胞进行重新编程，从而能够有助于不同身体部位的再生。
该研究小组发现在蝾螈和哺乳动物之间这一通路的主要差异，有助于我们理解为什么人类不能再生四肢，并揭示人类细胞的再生怎样改善。文章发表在《Stem Cell Reports 》期刊上。
研究证明ERK通路在哺乳动物细胞中并不十分活跃，但是当强迫其不断活动时，使得细胞更多可能性为重编程和再生。这可能有助于研究人员更好了解疾病并设计新疗法。
这项研究的*研究员，Max Yun 博士(伦敦大学学院结构与分子生物学)说：“虽然人类只有有限的再生能力，其他生物，如蝾螈，能够再生复杂结构的所有组成成分，包括它们的心脏、眼睛、脊髓、尾巴等部分，而且它们是*的成体脊椎动物能够再生出完整四肢的。”
我们很高兴发现一个重要的分子通路，ERK通路，它决定是否一个成体细胞能够重编程并帮助再生过程。操纵这一机制能促进定向治疗增强人细胞的再生可能性。
ERK通路是蛋白质从细胞表面传递信号到包含细胞遗传物质的细胞核中的一种方式。进一步研究将集中在了解这一重要途径在断肢再生中是如何调节的，以及相关过程中的其他分子。

美国

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【公司名称】 广州健仑生物科技有限公司
【】杨永汉
【】
【腾讯】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-3室
【企业文化】

In addition, PET / CT was also used during the experiment to measure the metabolic changes of brown fat, muscle and fat. Experimental results show that brown fat increases in cold months and falls in warm months.
One of the major benefits of increased brown fat mass is its increased sensitivity to insulin - meaning that people who have more brown fat in their body need less insulin to lower their blood glucose levels after a meal.
"Before this study, it was unknown whether there was any real control over the growth of brown fat in our body, and our study found that the cold months increased brown fat by 30% to 40%, while in the second month at 24 degrees Celsius, the brown Fat levels dropped back to baseline in April - the temperature was as high as 27 degrees and the amount of brown fat dropped below baseline. "
In Dr. Paul Lee's view, "Insulin sensitivity increases in tandem with the increase in brown fat, which in the future may open new avenues of treatment that will hinder sugar metabolism." On the other hand, people are being light due to rising heat in today's society Degrees of exposure to cold conditions are reduced, which may impair the function of brown fat, causing obesity and metabolic disorders. "
Studies have shown that in the United Kingdom in the past decades, the temperature in American family bedrooms, dining rooms and living rooms has risen from 19 degrees Celsius to 22 degrees Celsius, which will also lead to a decrease in brown fat production, so in addition to an unhealthy diet and lack of exercise, Exposure to increased temperatures at higher temperatures may also be one of the factors contributing to obesity. "
The secrets of successfully regenerating the body parts of the salamander are being uncovered, and researchers at the University College London apply it to humans. First, the researchers found that the 'ERK pathway' must be constantly reprogrammed for salamander cells to help regenerate different parts of the body.
The team found that the major differences in this pathway between salamander and mammals helped us to understand why humans can not regenerate their limbs and reveal how human cell regeneration can improve. Article published in "Stem Cell Reports" journal.
Studies have shown that the ERK pathway is not very active in mammalian cells, but when forced to continue its activity, the cells are more likely to be reprogrammed and regenerated. This may help researchers better understand the disease and design new therapies.
Dr. Max Yun, chief researcher of the study, Structural and Molecular Biology, University College London, said: "While humans have only limited ability to regenerate, other organisms, such as salamanders, are able to regenerate all the components of a complex structure, including their Heart, eyes, spinal cord, tail and other parts, and they are the only adult vertebrates capable of regenerating intact limbs. "
We are happy to find an important molecular pathway, the ERK pathway, that determines whether an adult cell can reprogram and help regenerate the process. Manipulating this mechanism can promote targeted therapy to enhance the regenerative potential of human cells.
The ERK pathway is one way in which proteins transfer signals from the cell surface into the nucleus containing cytogenetic material. Further research will focus on understanding how this important pathway is regulated in limb amputation and other molecules involved in the process.

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