编号	英文名称	中文名称
JL-FA-01	Amebiasis (AME)	阿米巴病
JL-FA-02	Allergens, Rast scores	过敏原，放射性过敏原吸收实验。指对特定的人群引起免疫反应或者过敏反应的食品中的蛋白质
JL-FA-03	Allergens, Rast scores negative	过敏原，放射性过敏原吸收实验阴性
JL-FA-04	Anti-cyclic citrullinated peptide Antibody (CCP) Arthritis	抗环瓜氨酸肽抗体
JL-FA-05	ASCA Saccharomyces Cerevi	人抗酿酒酵母抗体（ASCA）
JL-FA-06	Aspergillis	麴菌病
JL-FA-07	Beta 2 Glycoprotein	β2糖蛋白
JL-FA-08	Beta 2 Glycoprotein IgM	β2糖蛋白 IGM
JL-FA-09	Bordela Pertussis	百日咳杆菌
JL-FA-10	Bordela Pertussis IgM	百日咳杆菌 IGM
JL-FA-11	C-ANCA	C-抗中性粒细胞胞浆抗体（ANCA)
JL-FA-12	Cardiolipin	心肌磷脂
JL-FA-13	Cardiolipin IgA	心肌磷脂 IGA
JL-FA-14	Cardiolipin IgG	心肌磷脂 IGG
JL-FA-15	Cardiolipin IgM	心肌磷脂 IGM
JL-FA-16	Cerebral Spinal Fluid	脑脊髓液
JL-FA-17	Chagas	恰加斯病/南美锥虫
JL-FA-18	Chlamydia	衣原体
JL-FA-19	Chlamydia IgA	衣原体IGA
JL-FA-20	Chlamydia IgG	衣原体IGG
JL-FA-21	Chlamydia IgM	衣原体IGM
JL-FA-22	Chlamydia Neg	衣原体阴性
JL-FA-23	Clotting Factor C3	凝固因子C3
JL-FA-24	Clotting Factor C4	凝固因子C4
JL-FA-25	Coccidiodes	球孢菌
JL-FA-26	Cytomegalovirus (CMV) Neg	巨细胞病毒抗体阴性
JL-FA-27	CMV IgG	巨细胞病毒 IGG阳性
JL-FA-28	CMV IgM VCA	巨细胞病毒 IGM 阳性
JL-FA-29	C-Reactive Protein (CRP)	C-反应蛋白质
JL-FA-30	Dengue Fever	登革热
JL-FA-31	Dengue Fever IgM	登革热 IGM
JL-FA-32	DS (Double Stranded) DNA	双链脱氧核糖核酸
JL-FA-33	EBNA (Epstein-Barr nuclear antigen) IgG	EB病毒核抗原 IGG
JL-FA-34	EBNA (Epstein-Barr nuclear antigen) IgM	EB病毒核抗原 IGM
JL-FA-35	Epstein Barr Virus (EBV) Negative Plasma	EB病毒阴性血浆
JL-FA-36	Epstein Barr Virus (EBV) EA IgM	EB病毒早期抗原 IGM
JL-FA-37	Epstein Barr Virus (EBV) VCA IgM	EB病毒壳蛋白 IGM
JL-FA-38	Epstein Barr Virus (EBV) EA IgG	EB病毒早期抗原 IGG
JL-FA-39	EMA (Endomysial Antibodies)	肌内膜
JL-FA-40	Gliadin	麸蛋白,麦醇溶蛋白,麦胶蛋白
JL-FA-41	Gliadin IgG	麦醇溶蛋白 IGG
JL-FA-42	Gliadin IgA	麦醇溶蛋白 IGA
JL-FA-43	Glomerular Basement Membrane (GBMA)	肾小球基底膜病
JL-FA-44	Helicobacter pylori IgA	幽门螺旋杆菌IGA
JL-FA-45	Helicobacter pylori IgG	幽门螺旋杆菌IGG
JL-FA-46	Helicobacter pylori IgM	幽门螺旋杆菌IGM
JL-FA-47	Helicobacter pylori Negative	幽门螺旋杆菌阴性
JL-FA-48	Helicobacter pylori Positive Plasma	幽门螺旋杆菌阴性血浆
JL-FA-49	Hepatitis A Virus (HAV) Pos. Plasma	甲型肝炎病毒阳性血浆
JL-FA-50	Hepatitis A Virus (HAV) IgM	甲型肝炎病毒IGM
JL-FA-51	Hepatitis B Core (HBc) IgG	乙型肝炎病毒核心 IGG
JL-FA-52	Hepatitis B Core (HBc) IgM	乙型肝炎病毒核心 IGM
JL-FA-53	Anti Hbe (Antibody to HBV antigen)	乙肝抗体
JL-FA-54	Hepatitis Delta Virus	丁型肝炎病毒
JL-FA-55	HBeAg (HBV e antigen)	乙肝 E抗原
JL-FA-56	anti-HBs (HBV surface antibody)	乙肝表面抗体
JL-FA-57	Hepatitis B (HBsAg) "Chronic"	乙型肝炎（乙肝表面抗原）“慢性病
JL-FA-58	HBsAg (HBV surface antigen) Serum	乙肝表面抗原血清
JL-FA-59	HBsAg (AD)	乙肝表面抗原（AD）
JL-FA-60	HBsAg (AY)	乙肝表面抗原（AY）
JL-FA-61	HBV Positive Plasma	乙肝阳性血浆
JL-FA-62	HBV DNA Plasma	乙肝DNA血浆
JL-FA-63	HBV DNA Serum	乙肝DNA血清
JL-FA-64	HBV DNA type A	A型乙肝DNA
JL-FA-65	HBV DNA type B	B型乙肝DNA
JL-FA-66	HBV DNA type C	C型乙肝DNA
JL-FA-67	HBV DNA type D	D型乙肝DNA
JL-FA-68	HBV DNA type E	E型乙肝DNA
JL-FA-69	HBV DNA type F	F型乙肝DNA
JL-FA-70	HBV Antibody HCV Antibody Plasma CO-INFECTED	乙肝和丙肝联合感染血浆
JL-FA-71	HCV (Hepatitis C Virus) Antibody	丙型肝炎抗体
JL-FA-72	HCV Core Antigen Positive	丙肝核心抗原阳性
JL-FA-73	HCV RNA PLASMA Genotype 1	基因1型丙肝RNA 血浆
JL-FA-74	HCV RNA PLASMA Genotype 2	基因2型丙肝RNA 血浆
JL-FA-75	HCV RNA PLASMA Genotype 3	基因3型丙肝RNA 血浆
JL-FA-76	HCV RNA PLASMA Genotype 4	基因4型丙肝RNA 血浆
JL-FA-77	HCV RNA PLASMA Genotype 5	基因5型丙肝RNA 血浆
JL-FA-78	HCV RNA PLASMA Genotype 6	基因6型丙肝RNA 血浆
JL-FA-79	HCV Riba single band	丙肝免疫印迹单波段
JL-FA-80	HCV RIBA Pos. (multiple bands)	丙肝免疫印迹阳性多波段
JL-FA-81	HCV Negative	丙肝阴性
JL-FA-82	HCV RNA Pos (quantitative)	丙肝RNA阳性（定量）
JL-FA-83	Hepatitis E	戊型肝炎
JL-FA-84	Herpes Simplex Virus (HSV)1/2 Positive Plasma	单纯性疱疹病毒1/2阳性血浆
JL-FA-85	Herpes Simplex Virus (HSV) 1 Negative Plasma	单纯性疱疹病毒1 阴性血浆
JL-FA-86	Herpes Simplex Virus (HSV) 1 IgG	单纯性疱疹病毒1 IGG
JL-FA-87	Herpes Simplex Virus (HSV 1) IgM	单纯性疱疹病毒1 IGM
JL-FA-88	Herpes Simplex Virus (HSV) 2 IgG	单纯性疱疹病毒2 IGG
JL-FA-89	Herpes Simplex Virus (HSV) 2 IgM	单纯性疱疹病毒2 IGG
JL-FA-90	Histone	组蛋白
JL-FA-91	Human Anti Mouse Ab (HAMA)	人抗鼠抗体
JL-FA-92	Human immunodeficiency virus (HIV) 1 Neg	HIV I 阴性
JL-FA-93	anti Human immunodeficiency virus (HIV) 1 Plasma	抗HIV I 血浆
JL-FA-94	anti Human immunodeficiency virus (HIV) 1 Serum	抗HIV I 血清
JL-FA-95	anti Human immunodeficiency virus (HIV) 2 Western Blot Tested	抗HIV 2 免疫印迹
JL-FA-96	anti Human immunodeficiency virus (HIV) 1/2 2 HIV (+)	抗HIV 1/2 2 HIV阳性
JL-FA-97	Human immunodeficiency virus (HIV) Ag	HIV抗原
JL-FA-98	HIV RNA (quantitative) Plasma	HIV RNA 定量血浆
JL-FA-99	HIV RNA (quantitative) Serum	HIV RNA 定量血清
JL-FA-100	HIV1 Subtype A	HIV1 亚型A
JL-FA-101	HIV1 Subtype B	HIV1 亚型B
JL-FA-102	HIV1 Subtype C	HIV1 亚型C
JL-FA-103	HIV1 Subtype D	HIV1 亚型D
JL-FA-104	HIV1 Subtype E	HIV1 亚型E
JL-FA-105	HIV1 Subtype F	HIV1 亚型F
JL-FA-106	HIV1 Subtype G	HIV1 亚型G
JL-FA-107	HIV1 Subtype H	HIV1 亚型H
JL-FA-108	HIV1 Subtype J	HIV1 亚型J
JL-FA-109	HIV1 Subtype K	HIV1 亚型K
JL-FA-110	HIV1 Group O	HIV1 亚型O
JL-FA-111	Human immunodeficiency virus (HIV) 2 Antibody Plasma	HIV 2 抗体血浆
JL-FA-112	Human immunodeficiency virus (HIV) 2 Antibody Serum	HIV 2 抗体血清
JL-FA-113	HPV (Human Papiloma Virus) Negative	人乳状瘤病毒HPV阴性
JL-FA-114	HPV (Human Papiloma Virus) Positive	人乳状瘤病毒HPV阳性
JL-FA-115	Human immunodeficiency virus (HIV) Antibody HCV Antibody Plasma COINFECTED	HIV 抗体 HCV
JL-FA-116	Human T-cell Lymphotropic Virus (HTLV) I/II	人嗜T淋巴细胞病毒(HTLV) I/II
JL-FA-117	Human T-cell Lymphotropic Virus (HTLV) I	人嗜T淋巴细胞病毒(HTLV) I
JL-FA-118	Human T-cell Lymphotropic Virus (HTLV) II	人嗜T淋巴细胞病毒(HTLV) II
JL-FA-119	Jo-1	多发性肌炎抗原JO-1
JL-FA-120	IgE < 5,000 Ku/L	IgE < 5,000 Ku/L
JL-FA-121	Legionella	军团杆菌属
JL-FA-122	Leptospira	军团杆菌属
JL-FA-123	Lyme Disease	莱姆(氏)病:蜱传播的全身性疾病,常在夏季发生
JL-FA-124	Lyme IgG	莱姆(氏)病 IGG
JL-FA-125	Lyme IgM	莱姆(氏)病 IGM
JL-FA-126	Lyme Disease Neg	莱姆(氏)病阴性
JL-FA-127	Malaria	疟疾
JL-FA-128	Mononucleosis (infectious)	单核细胞增多症（有传染性的）
JL-FA-129	Mononucleosis Negative	单核细胞增多症阴性
JL-FA-130	Measles Negative	麻疹阴性
JL-FA-131	Measles IgG	麻疹 IGG
JL-FA-132	Measles IgM	麻疹 IGM
JL-FA-133	Microsomal Anti-thyroid peroxidase antibody (TPO) Positive Plasma Standard Titer (typically 1,000-3,000 IU/mL)	微粒体抗甲状腺过氧化物酶抗体
JL-FA-134	Microsomal Anti-thyroid peroxidase antibody (TPO) Negative Plasma	微粒体抗甲状腺过氧化物酶抗体
JL-FA-135	Anti-mitochondrial antibody (AMA)	抗线粒体抗体
JL-FA-136	Multiple Sclerosis	多发性硬化症
JL-FA-137	Mumps IgG	流行性腮腺炎 IGG
JL-FA-138	Mumps Ab IgM	流行性腮腺炎抗体 IGM
JL-FA-139	Mumps Antibody Negative Plasma	流行性腮腺炎抗体阴性血浆
JL-FA-140	Mumps Antibody Negative Serum	流行性腮腺炎抗体阴性血清
JL-FA-141	Myeloma Plasma	骨髓瘤血浆
JL-FA-142	Myeloma IgA	骨髓瘤IGA
JL-FA-143	Myeloma IgE	骨髓瘤IGE
JL-FA-144	Myeloma IgG	骨髓瘤IGG
JL-FA-145	Myeloma IgM	骨髓瘤IGM
JL-FA-146	Mycoplasma	支原体
JL-FA-147	Mycoplasma Negative	支原体阴性
JL-FA-148	Mycoplasma IgG	支原体IGG
JL-FA-149	Mycoplasma IgM	支原体IGM
JL-FA-150	Mycoplasma PCR	支原体PCR
JL-FA-151	Normal Human Plasma	正常人血浆
JL-FA-152	Normal Human Serum	正常人血清
JL-FA-153	Nuclear Antibody Centromere	核抗体着丝粒
JL-FA-154	Nuclear Antibody, Speckled ANA	核抗体，斑点抗核抗体
JL-FA-155	Nuclear Antibody, Nucleolar ANA	核抗体,核仁抗核抗体
JL-FA-156	Nuclear Antibody, Homogeneous ANA	核抗体，同质抗核抗体
JL-FA-157	Nuclear Antiobody, Speckled. (ANA) Negative	核抗体，斑点。抗核抗体阴性
JL-FA-158	P-ANCA (associated neutrophil cytoplasmic antibodies)	相关的嗜中性粒细胞胞浆抗体
JL-FA-159	Parietal Cell Antibody (PCA)	胃)壁细胞抗体
JL-FA-160	Parvo positive plasma	细小病毒阳性血浆
JL-FA-161	Parvo IgM	细小病毒 IGM
JL-FA-162	Parvo IgG	细小病毒 IGG
JL-FA-163	Parvo Negative Plasma	细小病毒阴性血浆
JL-FA-164	Parvo DNA positive	细小病毒 DNA 阳性
JL-FA-165	Phospholipid Positive Plasma	磷脂阳性血浆
JL-FA-166	Prothrombin	凝血酶原,凝血因子
JL-FA-167	Rheumatoid Factor (RF) <1000 IU/mL	类风湿因子<1000 IU/mL
JL-FA-168	Rheumatoid Factor (RF) 1001-2000 IU/mL	类风湿因子1001-2000 IU/mL
JL-FA-169	Rheumatoid Factor (RF) 2001-4000 IU/mL	类风湿因子 2001-4000 IU/mL
JL-FA-170	Rheumatoid Factor (RF) 4001-5000 IU/mL	类风湿因子 4001-5000 IU/mL
JL-FA-171	Rheumatoid Factor (RF) >5000 IU/mL	类风湿因子>5000 IU/mL
JL-FA-172	Ribonucleoprotein (RNP) Positive	核糖核蛋白阳性
JL-FA-173	Rubella Chimeric	风疹
JL-FA-174	Rubella Negative	风疹阴性
JL-FA-175	Rubella IgG	风疹IGG
JL-FA-176	Rubella IgM	风疹IGM
JL-FA-177	Rubeola Negative Plasma	风疹阴性血浆
JL-FA-178	Rubeola IgG	风疹IGG
JL-FA-179	Scleroderma (Scl-70) Pos	胶原沉着病,硬皮病,硬皮症阳性
JL-FA-180	Scleroderma (Scl-70) Negative	硬皮病阴性
JL-FA-181	Sickle Cell Fresh Whole Blood	镰刀形红细胞新鲜全血
JL-FA-182	Smith (SM)	抗Smith抗体阳性血清（SLE的特征性抗体）
JL-FA-183	SMITH RNP	抗RNP抗体阳性血清（SLE的特征性抗体）
JL-FA-184	Smooth Muscle (ASMA)	抗平滑肌抗体阳性血清
JL-FA-185	Sjogren syndrome antigen A (SSA) Positive	舍格伦综合征或干燥综合征抗原A 阳性
JL-FA-186	Sjogren syndrome antigen B (SSB) Positive	舍格伦综合征抗原B 阳性
JL-FA-187	Sjogren syndrome antigen B (SSB) Negative	舍格伦综合征抗原B阴性
JL-FA-188	Streptolysin O Ab (ASO)	链球菌溶血素O抗体
JL-FA-189	Syphilis (RPR - Rapid Plasma Reagin) Positive Plasma	梅毒（梅毒-快速血浆反应）阳性血浆
JL-FA-190	Syphilis (RPR - Rapid Plasma Reagin) Negative Plasma	梅毒（梅毒-快速血浆反应）阴性血浆
JL-FA-191	Syphilis/ATA/T. pallidum IgG	梅毒ATA/T，苍白球IGG
JL-FA-192	Syphilis/ATA/T. pallidum IgM	梅毒ATA/T，苍白球IGM
JL-FA-193	Systemic Lupus Erythematosus (SLE) Positive	全身性红斑狼疮阳性
JL-FA-194	Systemic Lupus Erythematosus (SLE) Negative	全身性红斑狼疮阴性
JL-FA-195	TG/TPO Positive (Standard Titer 1,000 - 3000 IU/mL)	甲状腺球蛋白/甲状腺过氧化物酶阳性
JL-FA-196	TG/TPO Negative	甲状腺球蛋白/甲状腺过氧化物酶阴性
JL-FA-197	TTG (Tissue Transglutaminase)	组织转谷氨酰胺酶
JL-FA-198	TTG (Tissue Transglutaminase) IgA	组织转谷氨酰胺酶 IGA
JL-FA-199	ToRCH (Toxo, Rubella, CMV, HSV) Positive	优生优育（弓形虫，风疹，巨细胞，单胞）阳性
JL-FA-200	ToRCH (Toxo, Rubella, CMV, HSV) Negative	优生优育（弓形虫，风疹，巨细胞，单胞）阴性
JL-FA-201	Toxoplasmosis (Toxo)	弓形虫病
JL-FA-202	Toxoplasmosis (Toxo) IgG	弓形虫病IGG
JL-FA-203	Toxoplasmosis (Toxo) IgM	弓形虫病IGM
JL-FA-204	Thyroglobulin (TG) Positive Plasma	甲状腺球蛋白阳性血浆
JL-FA-205	Thyroglobulin (TG) Negative	甲状腺球蛋白阴性
JL-FA-206	Varicella-Zoster Virus (VZV) Negative	水痘-带状疱疹病毒阴性
JL-FA-207	Varicella-Zoster Virus (VZV) IgG	水痘-带状疱疹病毒IGG
JL-FA-208	Varicella-Zoster Virus (VZV) IgM	水痘-带状疱疹病毒IGM
JL-FA-209	West Nile Virus (WNV)	西尼罗河脑炎病毒
JL-FA-210	West Nile Virus (WNV) IgM	西尼罗河脑炎病毒IGM

美国

干扰素伪装免疫细胞
免疫生物学教授Annette Oxenius的研究小组现在发现，是什么阻止nk细胞杀死免疫系统的“来自于其他部门的同事”：健康CD8 +T细胞能够检测免疫信使物质1型干扰素，1型干扰素通过与这些免疫细胞的特定表面受体结合，从而隐藏它们的应激。换句话说，1型干扰素作为一种伪装斗篷使得免疫细胞不被NK细胞看见。如果T细胞缺乏1型干扰素的停泊位点，那么它们就会被NK细胞追杀直至灭绝。
使用感染两种模型病毒的小鼠，研究人员发现，如果动物的CD8 +t细胞缺乏这些干扰素的受体，不仅NK细胞消除感染病毒的细胞，而且免疫细胞也被认为采取了行动，从而削弱了抗病毒的免疫反应。
训练有素的细胞死亡信号
研究人员使用CD8 + T细胞没有任何1型干扰素受体的小鼠探索工作机制是如何运作的，以及缺乏1型干扰素的CD8 + T细胞是如何耗尽NK细胞的。如果没有自然杀手的存在，尽管缺乏干扰素检测，但T细胞依然会增多，成熟和发育。此外，瑞士苏黎世联邦理工学院的免疫生物学家发现，这些无需传感器的T细胞逐渐形成其表面的“识别标签”，一旦与NK细胞接触，就会引发NK细胞的杀伤作用。因此，这些“应激标签”的表达通过干扰素结合以及通过T细胞上的干扰素受体发出信号被抑制。如果由于受体的缺乏而阻止了信号的传输，那么细胞会大量表达这些应激分子。
自身免疫性疾病的机制?
到目前为止，尚不清楚人类是否具有相同的机制。然而，人类免疫系统用来保护T细胞避免NK细胞攻击的基本过程很可能是类似的。在一方面，研究人员现在揭示应激T细胞需要保护自己免受NK细胞攻击的机制。另一方面，这项发现形成了新的假设。例如，可以想到的是，缺乏干扰素受体的T细胞活化显示出它们为“应激的”，并因此杀死了。自身免疫反应性T细胞的活化过程中可能会出现这样的情况，例如，它通常发生在不存在高浓度的1型干扰素。Oxenius和她的团队有很浓厚的兴趣将在未来几年内测试这些令人兴奋的假设。
加州大学圣地亚哥分校的生物学家发现能够使动物细胞产生核糖体的化学系统中的‘未知环节’——每个细胞中含有数千蛋白‘工厂’加工所有建立组织和维持生命所必须的蛋白。他们的发现不仅强迫分子生物学基础教科书的修订，而且也为科学家提供较好的理解如何限制自由的细胞生长，如癌症，也许通过控制核糖体的输出来调节。这一研究发现发表在6月23日的《Genes & Development》期刊上。

美国

二维码扫一扫

【公司名称】 广州健仑生物科技有限公司
【】杨永汉
【】
【腾讯】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-3室
【企业文化】

Interferon disguises immune cells
Annette Oxenius' team of immunobiology professors now finds out what "colleagues from other departments" are preventing nk cells from killing the immune system: Healthy CD8 + T cells are able to detect immunocompetent substances Type 1 interferon, type 1 interferon By binding to specific surface receptors of these immune cells, their stress is hidden. In other words, type 1 interferons act as a camouflage cape so that immune cells are not seen by NK cells. If T cells lack type 1 interferon docking sites, they are killed by NK cells until they become extinct.
Using mice infected with both model viruses, the researchers found that if the animal's CD8 + T cells lacked these interferon receptors, not only did NK cells eliminate the virus-infected cells, but immune cells were also thought to have taken action that weakened Antiviral immune response.
Trained cell death signal
Researchers using mice that did not have any Type 1 interferon receptor on CD8 + T cells explored how the working mechanism works and how NK cells are depleted by CD8 + T cells lacking type 1 interferon. Without the existence of a natural killer, T-cells will continue to proliferate, mature, and develop despite the lack of interferon testing. In addition, immune biologists at the Federal Institute of Technology in Zurich, Switzerland, found that these sensorless T cells evolved to form "labels" on their surface that, when contacted with NK cells, elicited NK cell killing. Thus, the expression of these "stress tags" is inhibited by interferon binding as well as signaling by interferon receptors on T cells. If signaling is blocked because of lack of receptors, the cells express large quantities of these stressors.
Mechanisms of autoimmune diseases?
So far, it is unclear whether human beings have the same mechanism. However, the basic process that the human immune system uses to protect T cells from NK cell challenge is likely to be similar. On the one hand, researchers now reveal that stressed T cells need a mechanism to protect themselves from attack by NK cells. On the other hand, this finding has led to new assumptions. For example, it is conceivable that T-cell activation, which lacks interferon receptors, shows that they are "stressed" and therefore killed. This may occur during the activation of autoimmune reactive T cells, for example, it typically occurs in the absence of high concentrations of type 1 interferons. Oxenius and her team are very interested in testing these exciting assumptions in the coming years.
Biologists at the University of California, San Diego discovered the 'unknown' part of the chemical system that produces ribosomes in animal cells - thousands of proteins in each cell. The 'factory' processes all the proteins necessary to build and sustain life. Their findings not only force the revision of basic textbooks in molecular biology, but also provide scientists with a better understanding of how to limit free cell growth, such as cancer, perhaps by controlling ribosomal output. The study was published in the June 23 issue of Genes & Development.

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